Whether you live in an apartment or a house, or you just want to keep your home free of EMFs, there are a number of methods to reduce exposure. One of the easiest is to reduce the use of electronic devices. You could also consider EMF block paint to prevent EMF radiation from entering your home. Another way to shield your house from EMF radiation is to install a shielding canopy for RF. It is a type made of net which contains EMF shielding and is used to prevent EMFs from entering a room. Another option is to have your home fitted with an enclosure that is conductive. They are referred to as Faraday cages.
Several studies have shown studies have shown that nonionizing EMF has antiproliferative properties in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed be based on the downregulation the cancer stem cell. This may account for the long-term responses seen in certain patients suffering from advanced HCC. But, the reason for AM EMF's impact on patients with cancer is not yet clear.
emf blockers from AM RF EMF on HCC tumour growth in vivo was studied in mice. The tumours were divided into 3 groups. The first group was not exposed to RF EMF. The second group was exposed RF EMF at a frequency that is similar to the one used by humans. In the third, they were subjected RF EMF with HCC-specific modulation frequencies. The effects of HCCMF on tumours was compared to that of RCF. The results showed that the cancers treated with HCCMF were significantly shrinking. However, the tumors treated with RCF didn't show evidence of tumour shrinkage.
The mechanism of tumor-specific AM RF EMF could be based on the fact that cancer cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect on HCC cells is caused through CACNA1H, a protein which mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H may have broader implications in the treatment and diagnosis of different cancers.
The tumours in those in the group that were unaffected to EMF from RF, and fed a standard mouse diet. The tumors in the HCCMF group were infected with Huh7 cells after they were five-seven weeks old. The tumors were removed when they showed excessive burden.
The tumors of the three groups showed distinct growth curves. The tumours treated with HCCMF had a significant reduction in tumour size after eight weeks. However, emf blocker treated with RCF didn't show shrinkage. The difference was highly significant. The tumors treated with RCF were able to show necrosis, which is common in tumours that are exposed to RCF. The possibility is that this necrosis was caused by a lack of oxygen in the more invasive tumours.
In summary, the results show an AM-RF EMF has anticancer properties in vitro as well as in the vivo. Numerous studies have demonstrated it is true that AM RF EMF produces measurable shrinkage of tumors for HCC patients. The possibility is that the AM EMF causes these effects because of CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may cause a lasting influence on the development of HCC tumors in the vivo.