If https://ctxt.io/2/AACQmYF0Fw reside in an apartment or a house or just want to keep your home free of EMFs, there are a number of ways you can reduce exposure. One of the easiest is to limit the use of electronic devices. It is also possible to use EMF blocker paint to block EMF radiation from entering your home. Another easy way to protect your house against EMF radiations is to put up a shielding canopy for RF. This is a fabric made of net which contains EMF shielding and is used to prevent EMFs from entering rooms. Another option is to have your home equipped with a conductive enclosure. They are referred to as Faraday cages.
Several studies have shown how the non-ionizing energy of RF can cause antiproliferative effects in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed involve down-regulation of cancer stem cells. emf neutralizer could be the reason for the long-term responses seen in some patients with advanced HCC. However, the mechanism of AM RF EMF's effect in cancer patients is not evident.
Aspects of AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were studied in mice. The tumours were divided into 3 groups. First, the group that was unaffected to RF EMF. Second group members were exposed to RF EMF at the same frequency to the frequency used by humans. In the third, they were subjected to RF EMF with HCC-specific modulation frequencies. The impact of HCCMF on tumours was compared to that of RCF. The results indicated that cancers treated with HCCMF were significantly shrinking. However, tumours treated with RCF showed no evidence of tumour shrinkage.
The reason for cancer-specific AM RF EMF could be due to the fact that cancer cells require Cav3*2 type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect in HCC cells is caused through CACNA1H, a protein which is responsible for the influx of Ca2+ specific to tumours. The results indicate that CACNA1H could have wider implications for treatment and diagnosis of many cancers.
The tumours in the control group were not exposed to EMF from radiofrequency, and fed a standard mouse diet. The tumours in HCCMF HCCMF group were injected with Huh7 cells when they were 5 to 7 weeks old. The tumours were then euthanized in cases of excessive burden.
The tumors of the three groups also showed distinct growth curves. The HCCMF-treated tumors showed a significant decrease in tumour size after eight weeks. However, tumors treated with RCF showed no reduction in size. The difference was highly significant. The tumors treated by RCF had necrosis, which is typical when tumors are that are exposed to RCF. There is emf radiation blocker that the necrosis was caused by the lack of oxygen in larger cancers.
In summary, the results suggest that AM RF EMF is a powerful source of anticancer activity in vitro and in vivo. Several studies have shown it is true that AM RF EMF produces measurable shrinkage of tumors within HCC patients. It is possible that AM RF EMF produces these effects through CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may exert a sustained impact on the development of HCC tumors in the vivo.