Whether you live in an apartment or a house, or you just would like to ensure your home is free from EMFs, there are a number of methods to reduce exposure. One of the easiest is to restrict your use of electronic devices. It is also possible to use EMF block paint to prevent EMF radiation from entering your home. Another way to shield your house from EMF radiations is to put up a shielding canopy for RF. This is a fabric made of net that has EMF shielding and is used to prevent EMFs from entering a room. Another option is to get your home fitted with a conductive enclosure. They are referred to as Faraday cages.
Several studies have shown how the non-ionizing energy of RF has antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed result from the deregulation of cancer stem cells. This could be the reason for the long-term responses seen in patients suffering from advanced HCC. However, the mechanism of AM EMF's effects on patients with cancer is not evident.
Aspects from AM electromagnetic fields (RFEM) on HCC tumour growth in vivo was studied in mice. The tumors were split into three groups. emf blocker was not exposed to RF EMF. Second group members were exposed RF EMF at a frequency that is similar to the frequency used by humans. The third group was exposed RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on the tumours was compared to that of RCF. emf blocking revealed that tumours treated with HCCMF showed significant shrinkage. However, the tumours treated with RCF didn't show evidence of shrinkage of the tumor.
The mechanism behind cancer-specific AM RF EMF could be due to the fact that tumor cells require Cav3*2 voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effect on HCC cells is mediated through CACNA1H, a protein which regulates the Ca2+ influx specific to tumors. emf blocker suggest that CACNA1H could have wider implications for the diagnosis and treatment of various cancers.
The tumors in the control group were not exposed to RF EMF, and were fed a standard mouse diet. The tumours in HCCMF HCCMF group were treated with Huh7 cells after they were 5 to 7 weeks old. The tumors were removed after they had a high burden.
The tumors of the three groups also displayed different growth curves. The tumors treated with HCCMF showed a significant decrease in size of the tumor after eight weeks. However, tumors treated with RCF showed no shrinkage. The difference was significant. The tumors treated with RCF showed necrosis that is common when tumors are that are exposed to RCF. The possibility is that the necrosis was caused by an absence of oxygen in larger tumors.
In sum, the results indicate an AM-RF EMF exhibits anticancer effects in vitro and in vivo. Several studies have shown that AM RF EMF produces measurable shrinkage of tumors for HCC patients. The possibility is that the AM EMF causes these effects because of CACNA1H which is a protein involved in tissue-specific Ca2+ influx. Additionally, AM RF EMF may cause a lasting impact on the development of HCC tumours in the vivo.